so more of my story. I am moving on from my addiction to sugar. empty stomach gastric hyperactivity disorder. sugar addiction sickness. sugar induced gastroparesis. I have figured out diabetes mellitus type 2 now.
so, yesterday I went on a date.
I met nicole.
I am helping with her health, and if she is really interested then I will keep assisting her.
before the date I prepared garlic, pear, apple, onion beef. a slice of shoulder of a cow in a puree of those fruits and vegetables and a very little bit of low sodium soy sauce and a korean barbeque sauce. it was great. I just had that one piece of meat and I felt satisfied and great. the meat was very lean with almost no fat on it.
the puree that i made for this meat and to use with other preparations and cookings was 1 entire garlic, 1 pear, 1 apple, and 1 white spanish onion. I blended and pureed these fruits and vegetables, and I also added 2 teaspoons of low sodium soy sauce and eight table spoons of a korean barbecue sauce.
the sauce is greater and is much healthier than using a lot of soy sauce, which is high in salt. even the low sodium soy sauce is high in salt.
this sauce or puree is also much healthier than using the korean barbecue sauce alone since that sauce is also high is salt and is not as fresh as the freshly pureed fruits and vegetables.
so that was great.
after the date I decided to test some sugary deserts on myself.
I chose to eat 1 slice of key lime pie. the key lime tasted great and still tastes great to me. it is even enjoyable to eat. but after one slice I do not really want more, but I am still not entirely satisfied. it is empty calories, i concede.
after that I had one serving of vanilla ice cream. the ice cream also tasted and felt great. the smooth, creamy texture and nice blend of sugar and milk, cream, eggs, and fat taste great. I was a little more satisfied, but I still wanted more on some level.
so, I had another serving of the ice cream. i had that second scoop. i felt great and rested and enjoy bliss again.
a few hours later I really just wanted to finish that last slice of pie in my fridge and so since I do not like to deny my cravings, I did.
that key lime pie has 45 grams of sugar per slice. 1/8 of that pie.
and the ice cream has 23 grams of sugar.
so I had at least 136 grams of sugar last night.
I also ate an entire bag of siete no salt chips with a guacamole salsa verde.
I knew I was over doing it on sugar especially, but I also knew that I was getting tired of most, if not all of these high sugar foods, and I was a little bit curious of how I would feel if I just ate as much as I was willing to.
so 2 slices of pie
2 scoops of vanilla ice cream
and 1 bag of siete chips, which is 5 servings of those no salt lower calorie chips.
I felt okay and went to bed. I slept well.
this morning though, I felt horrible.
I felt ill.
it was definitely the sugar, at the least, and also likely the no salt low calorie chips too. it was in part my over eating of these highly processed foods. when i over eat fruits and vegetables, I do not get these same ill effects, even though it is also unhealthy to over eat fruits and vegetables. but they are not as potent.
i had indigestion. i might have had a little bit of gerd, although it was not a lot of gerd. In the past I would not have diagnosed that little bit of sour taste at the back of my throat as gerd, but it is gerd, actually. gastroesophageal reflux disorder.
I was burping and the burping of 2 to 3 times was not settling my bloating and feelings of indigestion. I just kept trying to burp and burping and burped a lot.
my stomach felt like there was something in it and yet my stomach was not moving. it was not contracting and mixing and initiating digestion. but in fact, i am quite sure that I did not have any food in my stomach. I even tried to through up when the sensation to vomit hit me, but all that eventually came of my retching was spewing up some gastric acids and juices.
this is sugar addiction sickness.
i googled a set of terms describing my stomach discomfort: empty stomach gastric hyperactivity disorder.
and the results are at this link: https://www.google.com/search?q=empty+stomach+gastric+hyperactivity+disorder&rlz=1C1GCEU_enUS870US870&oq=empty+stomach+gastric+hyperactivity+disorder&aqs=chrome..69i57.4853j0j4&sourceid=chrome&ie=UTF-8
the main output from google, or search finding was gastroparesis.
Gastroparesis is a condition in which your stomach cannot empty itself of food in a normal fashion.https://www.webmd.com/digestive-disorders/digestive-disorders-gastroparesis#1
some more literature on gastroparesis that is important:
Abstract
ncbi.nlm.nih.gov/pubmed/18248660
And,
Corresponding author.
and indeed this is my gastroparesis. this is my sugar-induced gastroparesis.
so what was happening to me over night and this morning was sugar induced gastroparesis. which makes sense as gastroparesis is common in diabetes too.
"Poor control of blood sugar levels makes gastroparesis worse."Mar 30, 2019.
Signs and symptoms of gastroparesis include:
Some symptoms of gastroparesis
The list of gastroparesis symptoms as proposed by a contemporary review includes bloating, early satiety, nausea, postprandial fullness, and vomiting.1 Abdominal pain has also emerged as a common symptom.12-14 In a large multicenter study conducted by the National Institute of Diabetes and Digestive and Kidney Diseases (NIDDK), upper abdominal pain was rated as being moderate to severe by two-thirds of all patients surveyed and as the predominant symptom by one-fifth of all patients.14
Advances in Research of gastroparesis
Pioneering research points the way to the presence of fundamental pathologic findings in gastroparesis that may shed more light on the pathophysiology of gastroparesis symptoms and provide a more appropriate label (Figure).51-53 Employing full-thickness gastric biopsies, a loss of interstitial cells of Cajal (ICC) was identified as a common pathologic finding associated with delayed gastric emptying in diabetic, but not idiopathic, gastroparesis.51 However, ICC or enteric nerve loss did not correlate with symptom severity.51 In contrast, clinical severity and nausea were associated with a myenteric immune infiltrate among patients with idiopathic gastroparesis.51 The number of CD206-positive macrophages correlated with the number of ICC, suggesting that these macrophages may exert a protective effect on ICC in the human stomach.52 These studies offer hope for a future that is based on proven clinicopathologic correlations.
I met nicole.
I am helping with her health, and if she is really interested then I will keep assisting her.
before the date I prepared garlic, pear, apple, onion beef. a slice of shoulder of a cow in a puree of those fruits and vegetables and a very little bit of low sodium soy sauce and a korean barbeque sauce. it was great. I just had that one piece of meat and I felt satisfied and great. the meat was very lean with almost no fat on it.
the puree that i made for this meat and to use with other preparations and cookings was 1 entire garlic, 1 pear, 1 apple, and 1 white spanish onion. I blended and pureed these fruits and vegetables, and I also added 2 teaspoons of low sodium soy sauce and eight table spoons of a korean barbecue sauce.
the sauce is greater and is much healthier than using a lot of soy sauce, which is high in salt. even the low sodium soy sauce is high in salt.
this sauce or puree is also much healthier than using the korean barbecue sauce alone since that sauce is also high is salt and is not as fresh as the freshly pureed fruits and vegetables.
so that was great.
after the date I decided to test some sugary deserts on myself.
I chose to eat 1 slice of key lime pie. the key lime tasted great and still tastes great to me. it is even enjoyable to eat. but after one slice I do not really want more, but I am still not entirely satisfied. it is empty calories, i concede.
after that I had one serving of vanilla ice cream. the ice cream also tasted and felt great. the smooth, creamy texture and nice blend of sugar and milk, cream, eggs, and fat taste great. I was a little more satisfied, but I still wanted more on some level.
so, I had another serving of the ice cream. i had that second scoop. i felt great and rested and enjoy bliss again.
a few hours later I really just wanted to finish that last slice of pie in my fridge and so since I do not like to deny my cravings, I did.
that key lime pie has 45 grams of sugar per slice. 1/8 of that pie.
and the ice cream has 23 grams of sugar.
so I had at least 136 grams of sugar last night.
I also ate an entire bag of siete no salt chips with a guacamole salsa verde.
I knew I was over doing it on sugar especially, but I also knew that I was getting tired of most, if not all of these high sugar foods, and I was a little bit curious of how I would feel if I just ate as much as I was willing to.
so 2 slices of pie
2 scoops of vanilla ice cream
and 1 bag of siete chips, which is 5 servings of those no salt lower calorie chips.
I felt okay and went to bed. I slept well.
this morning though, I felt horrible.
I felt ill.
it was definitely the sugar, at the least, and also likely the no salt low calorie chips too. it was in part my over eating of these highly processed foods. when i over eat fruits and vegetables, I do not get these same ill effects, even though it is also unhealthy to over eat fruits and vegetables. but they are not as potent.
i had indigestion. i might have had a little bit of gerd, although it was not a lot of gerd. In the past I would not have diagnosed that little bit of sour taste at the back of my throat as gerd, but it is gerd, actually. gastroesophageal reflux disorder.
I was burping and the burping of 2 to 3 times was not settling my bloating and feelings of indigestion. I just kept trying to burp and burping and burped a lot.
my stomach felt like there was something in it and yet my stomach was not moving. it was not contracting and mixing and initiating digestion. but in fact, i am quite sure that I did not have any food in my stomach. I even tried to through up when the sensation to vomit hit me, but all that eventually came of my retching was spewing up some gastric acids and juices.
this is sugar addiction sickness.
i googled a set of terms describing my stomach discomfort: empty stomach gastric hyperactivity disorder.
and the results are at this link: https://www.google.com/search?q=empty+stomach+gastric+hyperactivity+disorder&rlz=1C1GCEU_enUS870US870&oq=empty+stomach+gastric+hyperactivity+disorder&aqs=chrome..69i57.4853j0j4&sourceid=chrome&ie=UTF-8
the main output from google, or search finding was gastroparesis.
Gastroparesis is a condition in which your stomach cannot empty itself of food in a normal fashion.https://www.webmd.com/digestive-disorders/digestive-disorders-gastroparesis#1
some more literature on gastroparesis that is important:
Aliment Pharmacol Ther. 2008 May;27(9):724-40. doi: 10.1111/j.1365-2036.2008.03637.x. Epub 2008 Feb 4.
Review article: gastroparesis.
Author information
- 1
- Centre for Gastroenterology, Royal Free Hampstead NHS Trust, London, UK.
Abstract
BACKGROUND:
Gastroparesis is a chronic disorder caused by stomach pump failure and characterized by profound nausea, vomiting and epigastric pain. Most often, the cause is unapparent and of the known associations, diabetes is the most common. Diagnosis is usually made using an isotope-labelled test meal. Treatment is incremental and includes education, dietary support, prokinetic and antiemetic agents. There are novel approaches including gastric neurostimulation.
AIM:
To review current concepts of gastric motor function, aetiology, investigation and treatment of gastroparesis.
METHODS:
A systematic web-based review of the literature was undertaken using a lexicon of terms associated with gastroparesis.
RESULTS:
There are few controlled studies of this condition. Little is known about causation or underlying nerve, muscle or pacemaker pathology. Idiopathic gastroparesis occurs most commonly in women and gastric emptying is often abnormal in diabetes. Isotopic gastric scintigraphy remains the gold standard investigation, but alternative tests are being developed. Treatment is multimodal and includes education, and nutritional support. There are no adequately powered controlled trials to support a particular drug regimen. In intractable gastroparesis, gastric neurostimulation appears to offer benefit.
CONCLUSION:
Despite a significant progress in the past decade, further controlled trials are required into the therapeutic options available for treating this intriguing condition.
- PMID:
- 18248660
- DOI:
- 10.1111/j.1365-2036.2008.03637.x
- [Indexed for MEDLINE]
ncbi.nlm.nih.gov/pubmed/18248660
And,
Gastroenterol Hepatol (N Y). 2018 Mar; 14(3): 140–145.
PMCID: PMC6004043
PMID: 29928159
A Critical Review of the Current Clinical Landscape of Gastroparesis
Corresponding author.
Address correspondence to: Dr Eamonn M. M. Quigley, 6550 Fannin St, SM 1201, Houston, TX 77030; E-mail: gro.tsidohtemnotsuoh@yelgiuqe
The authors have no relevant conflicts of interest to disclose.
Abstract
Gastroparesis has emerged as a common gastrointestinal disorder over the past few decades. It has been questioned whether this increase in prevalence reflects a true epidemic or rather the mislabeling of a variety of entities of similar symptomatology accompanied by a delay in the emptying of a meal from the stomach on a radionuclide gastric emptying study. Several factors contribute to this diagnostic morass, including a failure to recognize other conditions with similar symptoms, the relative convenience and accessibility of gastric emptying tests, the pervasive presence of some delay in gastric emptying in a variety of functional gastrointestinal disorders, and the confounding effects of certain therapies (opioids in particular) on gastric emptying rates. As a consequence, the label gastroparesis is affixed to patients whose symptoms have little to do with the rate at which food leaves the stomach and initiates a misdirected course of treatment that includes prokinetics, gastric electrical stimulation, and surgery. This strategy has already led to several well-documented therapeutic failures. When evaluating patients with upper gastrointestinal symptoms, the many facets of gastric and duodenal physiology that could contribute to symptoms should be considered, and a rush to attribute them to delayed gastric emptying should be resisted, as the subset of patients with accurately diagnosed gastroparesis is small. This opinion piece critically reviews the clinical landscape of gastroparesis as well as attempts to outline what should and should not be defined as clinically important gastroparesis.
and indeed this is my gastroparesis. this is my sugar-induced gastroparesis.
so what was happening to me over night and this morning was sugar induced gastroparesis. which makes sense as gastroparesis is common in diabetes too.
"Poor control of blood sugar levels makes gastroparesis worse."Mar 30, 2019.
Vomiting
Nausea
A feeling of fullness after eating just a few bites
Vomiting undigested food eaten a few hours earlier
Acid reflux
Abdominal bloating
Abdominal pain
Changes in blood sugar levels
Lack of appetite
Weight loss and malnutrition
Nausea
A feeling of fullness after eating just a few bites
Vomiting undigested food eaten a few hours earlier
Acid reflux
Abdominal bloating
Abdominal pain
Changes in blood sugar levels
Lack of appetite
Weight loss and malnutrition
so yeah. I now really know that over eating sugar is unhealthy and I have real symptomatology from it. it is not that this has happened to me before, because I have had gastroparesis before and I was even diagnosed with gastroparesis by peter massicot at harvard university health service when I was a medical student, but it did not seem so important then. I was not certain about the importance of this. his interaction with me made me think that maybe it just happens some times. I am quite sure it it was from my poor nutrition. and not just over eating, but also from eating poor foods. like refined foods with too much sugar.
Gastroparesis is defined as “a chronic symptomatic disorder of the stomach characterized by delayed emptying without evidence of mechanical obstruction.”1 Thus, 3 elements are central to the clinical diagnosis of gastroparesis: related symptoms, gastric emptying delay, and the absence of another organic explanation for the patient’s symptoms (eg, obstruction). Of these criteria, the last is the easiest to fulfill; endoscopy and a variety of imaging techniques can detect relevant diseases of the stomach and small intestine with considerable sensitivity and specificity. However, other questions remain: what symptoms should lead clinicians to suspect gastroparesis? How is its presence best defined? How relevant is gastric emptying delay to symptom pathogenesis? Is gastroparesis a valid target for therapeutic interventions? Although gastroparesis appears to be easily diagnosed with a gastric emptying test, the condition continues to present a challenge, with affected patients seeming to rapidly progress along a path of increasing levels of intervention without much improvement in outcome. Meanwhile, clinical investigators struggle to understand how a delay in the emptying of the solid and/or liquid components of a meal can explain a myriad of symptoms, and clinicians attempt to untangle the many factors that could lead to gastroparesis. To patients, this poorly understood condition could be debilitating, as it disrupts aspects of their personal and social lives. Gastroparesis is not uncommon; hospitalization rates for individuals in whom this condition was either a primary or secondary diagnosis increased significantly between 1995 and 2004,2 and it now represents a significant burden for health care systems due to the extent of health care utilization involved.1,3 A lack of specificity of symptoms attributed to gastroparesis (ie, early satiety, postprandial fullness, nausea, vomiting, upper abdominal pain1) and variations in the performance and interpretation of gastric emptying tests, together with a long history of therapeutic failures for drugs and other modalities designed to accelerate gastric emptying, call for a critical examination of the status of gastroparesis as a clinical entity.
Is gastroparesis a valid target for therapeutic interventions?
Yes! gastroparesis is a valid target for nutritional testing and nutritional education and nutritional modification. lifestyle changes. and this is very important for reducing disease, preventing worse diseases such as diabetes mellitus type 2, and also superiorizing one's health, nutrition, longevity, quality of life, and enjoyment.
so poor nutrition affects the stomach too. it is not just over eating at a meal. it is also the load on the stomach of those chemicals that is unhealthy. on the stomach and on the rest of the gastrointestinal system, and the rest of the body.
the stomach is an organ. no doubt about that and is not merely a bag that holds food.
Gastroparesis was first described among patients with type 1 diabetes and was typically complicated by end-organ complications such as nephropathy, retinopathy, and peripheral neuropathy.4
so I have figured out diabetes mellitus type 2 now. I know this disease well and it makes complete sense to me. it is a disease of nutrition. too much sugar in a human's diet.
https://jamwayconglomerate.wordpress.com/
https://jamwayconglomerate.wordpress.com/author/adriandkenny/
https://jamwayconglomerate.wordpress.com/author/adriandkenny/
If sugar remains in the bloodstream, the insulin-producing beta cells in the pancreas compensate by stepping up production. Eventually this leads to beta cell exhaustion, reduced insulin output and the appearance of full-blown diabetes.May 24, 2005
the Olmsted County study reports the incidence of gastroparesis among nondiabetic patients in the general population to be 1 in 1000 (0.1%).6
Additional studies show a preponderance of idiopathic gastroparesis among patients.8,9 Another study from Olmsted County suggests that the condition may have been underdiagnosed, noting a large discrepancy between the prevalence of diagnosed (by scintigraphy) gastroparesis (0.02%) and that of symptoms compatible with gastroparesis (1.8%).10
Outside of the aforementioned studies in which rigorous attention was paid to the definition of gastroparesis, it is our experience that the label of gastroparesis is loosely applied to a much broader and less clearly defined group of patients with unexplained nausea and vomiting.11 The (mis)diagnosis of this condition leads patients down a therapeutic path from dietary adjustments, prokinetics, antiemetics, pyloric botulinum toxin injections, placement of gastrostomy and jejunostomy tubes, and implantation of gastric electrical stimulators to various surgical approaches.
Some symptoms of gastroparesis
The list of gastroparesis symptoms as proposed by a contemporary review includes bloating, early satiety, nausea, postprandial fullness, and vomiting.1 Abdominal pain has also emerged as a common symptom.12-14 In a large multicenter study conducted by the National Institute of Diabetes and Digestive and Kidney Diseases (NIDDK), upper abdominal pain was rated as being moderate to severe by two-thirds of all patients surveyed and as the predominant symptom by one-fifth of all patients.14
Advances in Research of gastroparesis
Pioneering research points the way to the presence of fundamental pathologic findings in gastroparesis that may shed more light on the pathophysiology of gastroparesis symptoms and provide a more appropriate label (Figure).51-53 Employing full-thickness gastric biopsies, a loss of interstitial cells of Cajal (ICC) was identified as a common pathologic finding associated with delayed gastric emptying in diabetic, but not idiopathic, gastroparesis.51 However, ICC or enteric nerve loss did not correlate with symptom severity.51 In contrast, clinical severity and nausea were associated with a myenteric immune infiltrate among patients with idiopathic gastroparesis.51 The number of CD206-positive macrophages correlated with the number of ICC, suggesting that these macrophages may exert a protective effect on ICC in the human stomach.52 These studies offer hope for a future that is based on proven clinicopathologic correlations.
The pathophysiologic phenomena that contribute to symptoms in patients labeled as having gastroparesis may include:
Fundus fails to relax – loss of accommodation
Antral distension – if severe, could lead to pump failure
Pylorus “spasm” – recent studies suggest that loss of pyloric distensibility could cause a functional obstruction
Visceral hypersensitivity – a ubiquitous feature in many functional gastrointestinal disorders that could initiate or exacerbate symptoms
| Gastroenterol Hepatol (N Y). 2018 Mar; 14(3): 140–145. |
gastroparesis is one part of diabetes mellitus type 2. and occurs in high sugar diets. recurrent hyperglycemia.
anyway, I am not longer going to choose to keep having high sugar foods. instead I will have them occassionally or rather infrequently and opt for the other things that I enjoy and crave instead. I will continue to monitor how they make me feel whenever I ingest them.
adrian dane kenny, m.d.
jamway hospital
adrian.kenny@post.harvard.edu
617-697-0732
The diabetic stomach means a display of diabetic autonomic neuropathy. It is characterized by possibly debilitating gastrointestinal side effects and can likewise meddle with glucoregulation by adding to an endless loop of postponed purging of food or oral medications.
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